By Dra. Annabel Carungin
It is a syndrome with disordered metabolism and inappropriate hyperglycemia (increased blood sugar) due to either a deficiency of insulin secretion or to a combination of insulin resistance and inadequate insulin secretion to compensate.
It is generally divided into two categories: Type I, called insulin-dependent or juvenile diabetes and Type II, or non-insulin dependent diabetes or maturity-onset diabetes.
Type I diabetes is associated with destruction of the beta cells of the pancreas, which manufacture insulin. This type of diabetes occurs mostly in children and young adults. Recent evidence implicates a viral cause in some cases of this disorder. Autoimmune factors may also be involved.
Symptoms of type I DM include irritability, frequent urination, abnormal thirst, nausea or vomiting, weakness, fatigue, weight loss despite a normal (or even increased) intake of food and unusual hunger. In children, frequent bedwetting especially by a child who did hot previously wet the bed is another common sign.
People with type I diabetes are subject to episodes in which blood glucose levels are very high (hyperglycemia) and very low (hypoglycemia). Either of the conditions can lead to a serious medical emergency.
Episodes of hypoglycemia, which strike suddenly, can be caused by a missed meal, too much exercise, or a reaction to too much insulin. The initial signs of hypoglycemia are hunger, dizziness, sweating, confusion, palpitations and numbness of the lips. If not treated, the individual may go on to experience double vision, trembling and disorientation; may act strangely; and may eventually lapse into a coma.
In contrast, a hyperglycemic episode can come on over a period of several hours or even days. The risk for hyperglycemia is greatest during illness, when insulin requirements rise; blood sugar can creep up, ultimately resulting in coma, a reaction also known as Diabetic Ketoacidosis. One of the warning signs of developing hyperglycemia us the inability to keep down fluids.
Possible long term complications include stroke, blindness, heart disease, kidney failure, gangrene and nerve damage. The second category of DM, often referred to as adult or maturity onset diabetes, is most likely to occur in people with a family history of diabetes. In this type of disorder, the pancreas does produce insulin, but the insulin is ineffective. Symptoms include blurred vision, itching, unusual thirst, drowsiness, fatigue, skin infection, slow wound healing and tingling or numbness in the feet. The onset of type II diabetes
typically occurs during adulthood and is linked to a poor diet. Other signs that may be associated with diabetes include lingering flu-like symptoms, loss of hair on the legs, increased facial hair and small yellow bumps known as xanthomas anywhere on the body. Balanoposthitis (inflammation of the penis glands and foreskin) often is the first sign of diabetes and is usually associated with frequent urination day and night.
Some individuals have impaired glucose tolerance (IGT), indicating an asymptomatic, subclinical or latent form of diabetes. Studies indicate that there are 5 million adults with undetected type II diabetes and another 20 million have impaired glucose tolerance that may lead to full blown diabetes.
In summary, the Essentials of Diagnosis are:
Type I Diabetes:
- Polyuria, polydipsia and rapid weight loss associated with ransom plasma glucose > 200 mg/dL
- Plasma glucose of 126 mg/dL or higher after an overnight fast, documented on more than one occasion.
- Ketonemia, ketonuria or both.
Type II Diabetes:
Most patients are over 40 years of age and obese
- Polyuria and polydipsia. Ketonuria and weight loss generally are uncommon at time of diagnosis. Candidal vaginitis in women may be an initial manifestation. Many patients have few or no symptoms.
- Plasma glucose of 126 mg/dL or higher after an overnight fast on more than one occasion. After 75 g oral glucose and at least once between 0 and 2 hours.
- Hypertension, hyperlipidemia and atherosclerosis are often associated.
- Eat a high complex carbohydrate, low fat, high fiber diet including plenty of raw fruits and vegetables as well as fresh vegetable juices. This reduces the need for insulin and also lowers the level of fats in the blood. Fiber helps to reduce blood sugar surges. For snacks, eat oat or rice bran crackers with nut butter or cheese. Legumes, root vegetables and whole grains are also good.
- Other foods that help normalize blood sugar include berries, brewer's yeast, dairy products (especially cheese), egg yolks, fish, garlic, kelp, sauerkraut, soybeans and vegetables.
- Get your protein from vegetable sources such as grains and legumes. Fish and low-fat dairy products are also acceptable sources of protein.
- Avoid saturated fats and simple sugars (except when necessary to balance an insulin reaction).
- Eat more carbohydrates or reduce your insulin dosage before exercise. Exercise produces an insulin-like effect in the body. (Consult a doctor about the right approach for you.)
- Do not take fish/cod liver oil capsules or supplements containing large amount of para-aminobenzoic acid (PABA), and avoid salt and white flour products. Consumption of these products results in an elevation of blood sugar.
- Do not take supplements containing the amino acid cysteine. It has the ability to break down the bonds of the hormone insulin and interfere with absorption of insulin by the cells.
- Do not take extremely large doses of vitamins B1 (thiamine) and vitamin C (ascorbic acid). Excessive amounts may inactivate insulin.
- Do Biomagnetic Therapy
- Meridian Energizing Treatment for 10 minutes 3x a day for 1 week, then once a day for 5 weeks.
- Negative Super BioMagnet on right and left adrenal glands for 10 minutes. Then Positive Super Biomagnet on same locations for 10minutes. Then repeat the Negative.
- Negative Super BioMagnet on pancreas for 30-45 minutes, 2-3x daily.
- Drink Magnetized Negative water with MSO.